Any attempt to manually lengthen or stretch weak intrinsic and extrinsic spinal stabilizers causes greater forces to converge on the spinal discs and facets resulting in premature degeneration. When deterioration becomes severe it can be difficult to carry out the functions of daily living, let alone any high performance athletic endeavor. If the muscles, tendons, and fascia are relatively weak in relation to the demands of a particular sport or repetitive activity, the vertebral joints (usually beginning with the disc) will deteriorate (Fig 3). Due to reciprocal weakness, these folks tend to stand with the pelvis tilted one way or another with their butt kinda “hanging off” the hip capsules and lumbopelvic ligaments. ![]() ![]() It’s easy to spot those with weak glutes. The g-max and lat dorsi are not only dynamic lumbar spine stabilizers, but when working in conjunction with other spring systems, play a major role in coordinated cross-patterned gait (Fig 2). It attaches and is continuous with the biceps femoris, long dorsal SI ligaments, thoracolumbar fascia and crosses over to form the posterior spring system with latissimus dorsi (Fig 1). If reciprocal inhibition doesn’t completely rob its massive power, injured SI joints and low backs certainly will. Our flexion-addicted lame-ass (no pun intended) society had forced it to be neurologically bullied about by tight hip flexors. The gluteus maximus was probably the muscle most responsible for pulling us up onto two legs and now look what’s happened to it.
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